Post by omararias on Oct 23, 2015 10:59:02 GMT -6
Hi everyone,
I would like to post and discuss some information regarding the transportation of pararetroviruses during host infection, in this way we are gonna be correlating the classes topics about viral movement with specific examples. This is what I have found so far about this category of virures:
One of the most excelling examples of Pararetrovirus movement in the host are Caulimoviruses like the CaMV, reportedly this type of virus uses a MP (movement proteins) to facilitate and modify the structure of the plasmodesmata by insertion of tubular structures, in that way the viral particles can migrate from cell to cell.
Furthermore, during recent reports it has been highly suggested the role of different proteins in this mechanism, for instance protein products well known for exerting other functions like the P6 (formation of inclusion bodies) and VAP (virus associated protein responsible for replication and aphid transmission) might be associated with the transportation of virions from the sites of replication to modify plasmodesmata surfaces through coiled-coil domain interactions, and then to neighboring cells.
On the other hand, the modify plasmodesmata relate before result from an association between the viral MP and plasmodesmata host proteins like for example, calcium dependent target proteins, plasmodesmata localized Protein 1 –PDLP1 and MP17 in Arabidopsis thaliana. Therefore, this kind of mechanisms show a clear collaboration of a variety of viral proteins and host proteins and its importance in terms of communication throughout the plant (host).
Thanks.
Please if there is any question I would be more than happy to answer it.
I would like to post and discuss some information regarding the transportation of pararetroviruses during host infection, in this way we are gonna be correlating the classes topics about viral movement with specific examples. This is what I have found so far about this category of virures:
One of the most excelling examples of Pararetrovirus movement in the host are Caulimoviruses like the CaMV, reportedly this type of virus uses a MP (movement proteins) to facilitate and modify the structure of the plasmodesmata by insertion of tubular structures, in that way the viral particles can migrate from cell to cell.
Furthermore, during recent reports it has been highly suggested the role of different proteins in this mechanism, for instance protein products well known for exerting other functions like the P6 (formation of inclusion bodies) and VAP (virus associated protein responsible for replication and aphid transmission) might be associated with the transportation of virions from the sites of replication to modify plasmodesmata surfaces through coiled-coil domain interactions, and then to neighboring cells.
On the other hand, the modify plasmodesmata relate before result from an association between the viral MP and plasmodesmata host proteins like for example, calcium dependent target proteins, plasmodesmata localized Protein 1 –PDLP1 and MP17 in Arabidopsis thaliana. Therefore, this kind of mechanisms show a clear collaboration of a variety of viral proteins and host proteins and its importance in terms of communication throughout the plant (host).
Thanks.
Please if there is any question I would be more than happy to answer it.